I would like to have the ability to connect my Misfit Shine with my wellness apps from my physician and upload and track my progress on my aforementioned goals of fitness, nutrition and mental clarity (This one is the biggest stretch, I think.It would be great to have access to personalized information based on my medical history with reminders, goals, and recommendations in a quick, usable space and on any portable device I choose (Enter Portal, mobile apps, wellness management, and possibly even predictive analytics).I would like to have all of my healthcare records in one place, not scattered around in two different Health Systems and multiple disconnected physician’s offices (Enter HIE, portal, “Blue Button”, PHI).Hard to start the year that way! So here is how I would envision MY personal healthcare, if I had control: As I look forward in this New Year, one of my current goals is unobtainable. I would suspect that this will change with the imminent arrival of Meaningful Use Stage 2. © 2000 John Wiley & Sons, Inc.In a world where technology is rapidly advancing and user expectations are rising, it’s no longer enough to have an average user experience to delight your users and surpass your competition you must strive for the exceptional.Ĭurrently, my physician’s office is not connected. These mechanisms of paresthesia generation can account for paresthesias in normal subjects and may be relevant in some peripheral nerve disorders. The electrochemical gradient for K+ is reversed, and inward K+ currents trigger regenerative depolarization. Postischemic and posttetanic paresthesias occur when hyperpolarization by the Na+/K+ pump is transiently prevented by raised extracellular K+. The alkaline shift produced by hyperventilation selectively increases the persistent Na+ conductance, while the membrane depolarization produced by ischemia affects both transient and persistent Na+ channels. Ectopic discharges can be induced in normal afferents by four maneuvers: hyperventilation, ischemia, release of ischemia, and prolonged tetanization. These differences probably include more persistent Na+ conductance and inward rectification on cutaneous afferents, properties which probably confer greater protection from impulse‐dependent conduction failure but create a greater tendency to ectopic activity. Cutaneous afferents are more excitable than motor axons, due to differences in their biophysical properties. Paresthesias are common manifestations of central and peripheral pathological processes and are due to ectopic impulse activity in cutaneous afferents or their central projections. Mogyoros, Ilona Bostock, Hugh Burke, David Mechanisms of paresthesias arising from healthy axons Mechanisms of paresthesias arising from healthy axons
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